CardioMotief CD-Rom serie

Middels het doornemen van het CD-Rom-programma CardioMotief en het beantwoorden van de bijbehorende toetsvragen kunt u per CardioMotief uitgave 1 accreditatiepunt verwerven.

Klik hier om naar de toetsvragen van de actuele uitgave van CardioMotief te gaan.

Klik hier om naar de toetsvragen van eerdere uitgaven van CardioMotief te gaan.

U kunt hier de pre-assessment en post study toetsvragen beantwoorden behorend bij de Cardiovascular Self Study Series van de American Heart Association. U kunt de post study toets alleen beantwoorden indien u de pre-assessment toets van het betreffende deel reeds heeft voldaan.

Per deel heeft u na juiste beantwoording recht op 1 accreditatiepunt (totaal 3).

Let op: u kunt de vragen slechts één keer beantwoorden. Feedback m.b.t. het resultaat volgt na de post study toets.

Indien u de toewijzing van uw accreditatiepunten wenst te bestendigen logt u na het beantwoorden van de vragen in, dit scherm dient zich aan nadat u op "Verstuur" heeft geklikt. De punten zullen dan op uw conto worden bijgeschreven.

Deel 1 Atherotrombotic Cardiovascular Disease: Assessment & Management - Disease Burden & Pathophysiology

» Pre-Assessement

» Post Study

Deel 2 Atherotrombotic Cardiovascular Disease: Assessment & Management - Diagnostic Aspects

» Pre-Assessement

» Post Study

Deel 3 Atherotrombotic Cardiovascular Disease: Assessment & Management - Prevention & Treatment

» Pre-Assessement

» Post Study

Klik hier om uw resultaten op te vragen.

 

1. This is true of cholesterol that is linked to high-density lipoproteins (HDL):

Its increase is considered to be an atherosclerotic vasculopathy protector factor.

Its decrease is one of the components that make up the metabolic syndrome.

Following menopause the levels usually decrease.

All of the above are true.

All of the above are false.

2. The following can be considered a diagnostic sign of subclinical atherosclerosis:

Arterovenous crossing in the back of the eye.

Ankle/arm index of < 0.9.

Increase of left ventricular mass index.

Elevated C-reactive protein concentration in blood.

Abnormal ECG results following cardiac stress test.

3. Which of the following histological characteristics is true of vulnerable atherosclerotic plaque?

Endothelial dysfunction.

Low cholesterol content.

Presence of large quantities of monocytesmacrophages.

Presence of large quantities of smooth muscle cells.

Thick fibrous capsule.

4. Which of the following are the three most prevalent atherosclerosis risk factors according to the REACH registry?

Arterial hypertension, hypercholestoremia, and diabetes.

Hypercholestoremia, diabetes, and tabaquism.

Arterial hypertension, diabetes, and overweightobesity.

Hypercholestoremia, diabetes, and overweightobesity.

Diabetes, overweight-obesity, and tabaquism.

5. Which of the following individuals could be considered to be at high risk for developing an atherothrombotic complication in the next 10 years?

35 year-old male with diabetes.

50 year-old female smoker with an increase in carotid intima-media thickness.

70 year-old make with 168/62 mm Hg arterial pressure.

All of the above.

None of the above.

6. Which of the following form part of atherothrombotic disease?

Abdominal aorta aneurysm.

Auricular fibrillation.

Heart failure.

All of the above.

None of the above.

7. Acute coronary complications are usually triggered by which of the following actions:

Thrombomodulin.

Thrombin.

The tissular factor.

Fibrinogen.

Factor Xa.

8. The principal mechanism by which low-density lipoprotein (LDL)-cholesterol intervenes during genesis of atheroma plaque is its deposition in which of the following:

Adventitia.

In the artery middle layer.

Subendothelial.

Inside endothelial cells.

In the endothelium?s outer surface.

9. The principal mechanism by which LDL-cholesterol intervenes during atheroma plaque complication is:

By forming part of the atheroma plaque?s central nucleus.

By stimulating smooth-muscle cell proliferation.

By recruiting circular platelets at the site of the plaque.

By forming part of foamy cells.

By inducing thrombosis on the plaque interior.

10. Atherothrombotic disease appears:

In different territories depending on the each particular patient.

Frequently in several territories within the same patient.

Mostly in the peripheral layer when smokers are concerned, and in the coronary when dyslipidemia is concerned.

Always in the coronary-vasculocerebral-peripheral chronological order.

In all territories, but is more serious the higher the cholesterol level.